These findings support the concept that sodium-driven clearance of alveolar fluid may have a pathogenic role in pulmonary edema in humans and therefore represent an appropriate target for therapy.

Edema may not be visible until the amount of lung water increases by 30 percent. 33 Also, any radiolucent material that fills the air spaces (such as alveolar hemorrhage, pus, and bronchoalveolar ...

Alternatively, research suggests that mechanisms such as increased pulmonary hydrostatic pressure caused by enhanced venous return, pressure-induced mechanical disruption of the alveolar capillaries, ...

Gene therapy could be used as a targeted treatment for pulmonary edema. Delivery of specific genes into lung tissues stops the lung membrane leakiness, thereby reducing the progression of ...

video: Fluorescent fibrin clots in the alveolar microchannel of the human lung-on-a-chip. This image relates to a paper that appeared in the Nov. 7, 2012, issue of Science Translational Medicine ...

Lung macrophages play a pivotal role in diseases like idiopathic pulmonary fibrosis. Two types of macrophages—the white blood ...

More information: James B. Grotberg et al, Computational pulmonary edema: A microvascular model of alveolar capillary and interstitial flow, APL Bioengineering (2022).

However, in postobstructive pulmonary edema, β-agonists may accelerate the rate of alveolar fluid clearance and facilitate regression of pulmonary edema via increased active cation transport. 12 ...

If Pulmonary oedema is untreated it will increase pressure in the pulmonary artery leading to a weak right ventricle leading to further complications- 1. Pleural effusion.

In a study of 53 patients with acute cardiogenic pulmonary edema, injury of the alveolar–capillary barrier was associated with increased levels of plasma pulmonary surfactant-associated proteins ...